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naraburns  nihil supernum  1yr ago (text post)   32051 thread views

Culture War Roundup for the week of October 31, 2022

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A few days ago, I made a comment defending Turkheimer and critizing HBDers, in response to a vaulted "best of" comment dismissing Turkheimer. One of the main things my comment centered on was the phenotypic null hypothesis, which can roughly speaking be summarized as "correlation does not imply confounding" + "causation does not imply unmediated and unmoderated causation". Or as I phrased it:

Put simply, the phenotypic null hypothesis is this: Heritability tells you that if you go up through the chain of causation, then you will often end up with genes. However, there may be many ways that variables can be connected to each other, and there’s no particular reason to expect that every step along the chain of causation from genes to outcomes is best thought of as biological.

The consensus claimed that this was well-understood by HBDers around here, and perhaps even by HBDers more generally. Now I don't know that I buy that because it really doesn't seem well-understood in many places other than with people around Turkheimer.

In the thread, one person ended up posting an example of a paper which supposedly understood the nuances I was talking about. However, I disagree with that, and think that it is instead an excellent example of the problems with HBD epistemics. For instance, the paper opens by saying that the goal is to test evolutionary psychology hypotheses by testing for heritability in some personality traits:

According to the recent evolutionary-inspired theories (i.e., differential susceptibility [1], biological sensitivity to context [2]), humans, like many other species [3], differ substantially in their sensitivity to contextual factors, with some more susceptible to environmental influences than others. Importantly, these theories suggest that heightened sensitivity predicts both the reactivity to adverse contexts as well as the propensity to benefit from supportive features of positive environments. In other words, sensitivity is proposed to influence the impact of environmental influences in a “for better and for worse” manner [4]. These prominent theories converge on the proposition that genetic factors play a significant role in individual differences in Environmental Sensitivity (ES) [1, 2, 5].

Now, if you don't appreciate the phenotypic null hypothesis, then this would probably seem like a reasonable or even excellent idea. Evolution is about how genes are selected based on the traits they produce; if something is genetically coded, then evolution must have produced it, and conversely if evolution has produced it then it must be genetic.

But if you appreciate the phenotypic null hypothesis, then this study is of minimal relevance, almost no evidentiary value, and perhaps even eye-rollingly stupid. Of course the scales you administer are going to be heritable, because pretty much everything is heritable. Heritability doesn't mean that you've got anything meaningfully biological.

Now the thing is, my impression is that behavior geneticists do this sort of nonsense all the time, and that HBDers take them seriously when they do it. If HBDers instead properly appreciated the phenotypic null hypothesis, they would look somewhere else for this sort of info, or maybe even fix behavior genetics by propagating the info backwards to HBD-sympathetic behavior geneticists that they should read more Turkheimer. Notably, since this study was suggested as exemplary by someone here on TheMotte, it seems to provide at least an existence proof of someone who does not have a proper understanding of the phenotypic null hypothesis.

I appreciate you trying to bring nuance to the conversation, but without some examples it's still not clear to me what sorts of things you disagree with HBDers about exactly. I think the most relevant question is the extent to which the gaps in intellectual achievement, employment in various professions, and crime rates could realistically be changed by policy interventions. Do you think you have a much different answer here than HBDers?

As far as I can tell, you're saying that heritable traits might be caused along the way by others treating people differently based on their phenotype, and if that differential treatment were to go away, the presumably so would the heritability. Is that a fair summary?

And as an aside, I find the name "phenotypic null hypothesis" to be a bad name for two reasons: 1) It's not descriptive, and 2) it seems to be playing a rhetorical game by calling itself the "null hypothesis". I prefer to discuss evidence for and against various claims rather than arguing about who has the burden of proof.

I appreciate you trying to bring nuance to the conversation, but without some examples it's still not clear to me what sorts of things you disagree with HBDers about exactly.

I gave an example? The linked study? It's absolutely absurd methodology for testing the stated research question.

I think the most relevant question is the extent to which the gaps in intellectual achievement, employment in various professions, and crime rates could realistically be changed by policy interventions.

There are many possible questions one can come up with, and this is one of them, but it's not the one I'm making the threads about.

I'm making the threads about the phenotypic null hypothesis, which is a critical question for how to interpret various other kinds of evidence like twin studies.

As far as I can tell, you're saying that heritable traits might be caused along the way by others treating people differently based on their phenotype, and if that differential treatment were to go away, the presumably so would the heritability. Is that a fair summary?

Yes that is part of it, for instance if you are smart due to genetics then people will give you more education credentials and more money, and if instead people gave everyone a random amount of money, the heritability of income would disappear.

However it doesn't technically speaking have to be others treatment. It might also be your own treatment, or a result of other things like disease or physics or lots of things. Basically, variables affect each other across different levels, and that prevents heritability from distinguishing biological from nonbiological levels.

And as an aside, I find the name "phenotypic null hypothesis" to be a bad name for two reasons: 1) It's not descriptive, and 2) it seems to be playing a rhetorical game by calling itself the "null hypothesis". I prefer to discuss evidence for and against various claims rather than arguing about who has the burden of proof.

I think it has a good reason to call itself the null hypothesis. It would be absurd to claim that the heritability of income or marriage was not due to the way other people treat you phenotypically. You can't have a serious debate about HBD without the phenotypic null hypothesis being properly understood.

But so what if it is due to people treating you phenotypically? This is still genetic causation. It might indeed be interesting to some to figure out exactly through what mechanisms differences in genes result differences in income, but how exactly is this relevant for our ability to predict real world outcome of our policies? Can you show one example where “naive” (according to you) HBD would get some real world application seriously wrong, compared to approach informed by your phenotypic casual pathway correction?

I'm not talking about how things morally ought to be, I'm trying to figure out how the world is actually like. For the purpose of e.g. doing evolutionary psychology, such as with the linked study, it is relevant to know that heritability is of no evidentiary value.

it is relevant to know that heritability is of no evidentiary value.

You have not shown this. You've merely shown that heritability is not 100% ironclad.

I await your answer as to which concrete predictions HBDers will get wrong due to neglect of this.

Dude, read a few twin studies, they find that pretty much everything is heritable, of course heritability is of no evidentiary value.

This is probably the lousiest attempt at making an argument that I have seen here. The only way you could have made it worse is if you waved a credential.

I don't need to attempt hard to make an argument if the issue is very straightforward. If P(B) is high then P(A|B) ~ P(A).

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