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Are puberty blockers chemical castration?
A follow-up to the discussion with @netstack
This was originally a deep-chain reply, but after a few spergy, reddit-tier replies on my end, and @netstack's saintly curiosity, the conversation resulted in a decent-quality argument, that I'd like to get more eyes on and see I missed any obvious objections.
I did some extra digging as well. The wiki for Lupron links to the paper "Reforming (purportedly) Non-Punitive Responses to Sexual Offending", and while it's about triptorelin instead of Lupron, it's another GnRH. In any case a systematic review of the use of GnRH on sexual offenders (sci-hub) should hopefully settle the matter.
As a side note this paper makes me think the difference between GnRH's and DMPA's is that the former have (or promised to have) fewer side effects, not that they work on a fundamentally different principle (and while we're on the subject, let me just say I'm rather bemused at all the handwringing in all these papers about the side effects of these drugs on convicted sex offenders, when I compare them to the dismissal of any such concerns around giving the same drugs to children).
No, it just completely went over my head, lol.
This is a fun one. From what I understand chemical castration is meant to be reversible. This is what the wiki for chemical castration says right on the top, and I saw, but failed to bookmark, a paper that made that claim about DMPA's specifically, but that seems to be the general consensus on chemical castration:
So if irreversibility is a necessary condition for classifying something as chemical castration... than it seems that chemical castration does not meet the standard.
Now, I'm somewhat sympathetic to the "non-central" argument, you can argue that something that's reversible doesn't quite have that quality of having one's balls cut off that you'd expect from a term like "castration". It is also true you're going to have a hard time finding sources about the reversibility of puberty blockers, since dr. Cass' team looked, and all they can say is:
But when gender care providers themselves tell me that "puberty blockers are reversible (asterisk)", the asterisk being you can't stay on them too long, or that if you start them too early you're never going to have an orgasm, when celebrity cases like Jazz Jennings say they don't regret going on blockers, but the downside was "there wasn't enough tissue to work with when it came to the surgery" (and also don't know what an orgasm is), when the industry comes up with procedures like sigmoid vaginoplasties or zero depth vaginoplasties to either hack around or throw up their hands about the issue, can we say that there are good reasons to suspect some of the changes may be irreversible? What is even supposed to be the mechanism for reversibility? For a fully developed adult it's just a question of restoring testosterone levels and sperm counts, but for a child that never went through puberty we're basically hoping their body will catch up with development as if nothing ever happened.
Yeah, I know that as far as evidence goes, this doesn't rise to the standard of a proper well-designed study, but like I said in the other comment, the gender industry isn't particularly transparent about results they don't like. I understand wanting to remain agnostic on the reversibility question, but if you grant that these concerns are reasonable, it seems like puberty blockers are an at least as, and may possibly turn out to be more of, a central example of chemical castration, than chemical castration itself.
It depends how you define castration. The strict definition would be a double orchiectomy. If these chemicals made your balls wither up and drop off then yes, that plainly qualifies as chemically induced castration.
It feels like the original chemical castration usage must have arisen as a way to square the demands to castrate sex offenders with a means to backtrack in the face of appeals or wrongful convictions and preserve human rights: We'll castrate them [permanently] and any objections are moot because if we get it wrong it's totally reversible [and not really castration].
If you define it as anything that reduces normal sexual function then you put it on a vague and very wide spectrum and it becomes a matter of arguing the balance. The trouble is that would drag a lot of other things into the category. Too much whisky? Recreational amphetamines? SSRIs? It's starting to look like I've been chemically castrated a few times and it reversed rapidly with a good night's sleep and some eggs and coffee. What looked like a powerful rhetorical weapon to attack the trans movement finds itself a little impotent.
What if you carefully constructed a definition that captures the trans youth movement but leaves clinically depressed fans of Lemmy Kilmister unaffected? Well then it just looks like you're playing your own version of the "things are what they are because I said so" game.
If you think puberty blockers are bad because they have irreversible negative effects on fertility and sexual function then you can make that argument without the need for hyperbole.
Even if you're right about the origins of the term, it is a simple fact that the term was used in academic / law-enforcement literature, and no one seemed to object. I'm merely asking if puberty blockers fit into that previously-used-without-objection definition. My conclusion is: yes. Do you disagree?
I'm using the term in the exact same way it was used before puberty blockers entered public discourse, and even allowing for some stricter criteria that would stem from the discrepancy between the technical and colloquial terms. If this is hyperbole, every academic who has ever used the term was being hyperbolic.
I don't agree with the definition. It would classify a child being prescribed puberty blockers as an on-label treatment for precocious puberty as being chemically castrated.
I don't really see the issue, don't we already say that about cancer treatments? "We have to chemically castrate you to fight your cancer" sounds like a concise way of describing the costs and benefits of the treatment.
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